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Resistance to acyclovir, which is mediated by the viral TK, occurs by three mechanisms: (1) selection of a TK-deficient mutant; (2) selection of a TK-low producer mutant of herpes simplex; (3) selection of a mutant that produces an altered TK which is capable of phosphorylation of thymidine but no longer phosphorylates acyclovir (7, 8).

A sim- ilar ring of negative charges lining the cytosolic pore surface also helps select cations for passage (see Figure 7-45). In lieu of surgery or autopsy, a definitive diagnosis can be established by demonstrating (1) a characteristic vegetation, valve ring abscess, or new prosthetic valve dehiscence with echocardiography and (2) intravascular infection with multiple blood cultures obtained over an extended period that are positive for a microorganism consistent with endocarditis.

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