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1266 See the information section on general monographs (cover pages) Mechanisms of Psychostimulant Abuse Using Knockout Mice 95 95 Table derivativf Principal Observations Concerning Dopamine Receptor Null Mice After Psychostimulant Drug Treatmenta D1 receptor null mice Cocaine: no induction of locomotion and stereotypes Cocaine: no locomotor sensitization and no conditioned place preference D5 receptor null mice D2 receptor null mice No cAMP pathway stimulation Elevated midbrain dopamine levels Hyperactivity Reduced level of anxiety ND ND ND D3 receptor null mice Cocaine and amphetamine: supersensitivity to locomotor and stereotyped effects Cocaine and amphetamine: facilitated place preference D4 receptor null mice Cocaine and amphetamine: supersensitivity to locomotor effects ND Basal characteristics Hyperactivity (59) Acute exposure to psychostimulants Chronic exposures to psychostimulants Derivvative (60) Disorganization of the striatal complex Amphetamine: attenuated induction derivative trading in india ppt locomotion Amphetamine: no locomotor sensitization (68) or moderate locomotor sensitization (29) Severe neurological impairments (75) Cocaine: Lowered locomotor function (unpublished data) Modest neurological impairments pppt Loss of autoreceptor function Elevated enkephalin levels Decrease in DAT function Hyperactivity ND ND Elevated striatal dopamine levels Hypoactivity Cocaine: heightened c-fos and dynorphin levels Cocaine: heightened expression of neurotensin Reduced response to novelty Increased rotarod performances Elevated midbrain dopamine levels ND not determined.
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